tags:
Foundation is
A, neutrophil-activating
B, calcium overload
C, vascular endothelial cell swelling
D, ATP reduced
E, microcirculation blood flow is slow
Answer: A point where there is doubt
Resolution: 194 534. [pathophysiological Pok] cardiac ischemia-reperfusion injury following change is incorrect
A, myocardial systolic and diastolic dysfunction
B, ATP reduced
C, arrhythmia
D, phosphorus acid, creatine increased
E, myocardial ultrastructural injury
Answer: D have a question here
resolution:
194535. [pathophysiological Pok] following reperfusion was found to be inappropriate
A, low
B, low temperature
C, low pH
D, calcium
E, low-magnesium
Answer: E have questions here
Resolution: 194 536. [pathophysiological Pok] heart failure, the most characteristic changes in hemodynamic Pok
A, pulmonary circulatory congestion
B, arterial blood pressure
C, reduced cardiac output
D, increased precapillary resistance
E, systemic venous congestion
Answer: C There are questions here
resolution:
194537. [pathophysiological Pok] the following diseases can cause low output heart failure is
A, hyperthyroidism
B, severe anemia
C, myocardial infarction
D, beriberi (VitB lack of)
E, dynamic – arteriovenous fistula
Answer: C There are questions here
Resolution: 194 538. [pathophysiological Pok] The following are the characteristics of cardiac concentric hypertrophy is
A, increase muscle fiber length
B, hyperplasia of myocardial fibers were parallel
C, the heart chamber to expand
D, wall thickening is not obvious
E, Room chamber diameter and wall thickness ratio greater than normal
Answer: B here in doubt
resolution:
194539. [pathophysiological Pok] the following diseases can cause increased left ventricular afterload is
A, hyperthyroidism
B, severe anemia
C, myocarditis
D, myocardial infarction
E, hypertension
Answer: E have questions here
Resolution: 194 600. [pathophysiological Pok] The following does not belong to the duration of shock is changes in the microcirculation
A, arterioles, arteriolar contraction after
B, move – contraction of venous anastomosis
C, precapillary sphincter contraction
D, true capillaries close
E, less filling less flow, less flow irrigation
Answer: B here in doubt
resolution:
194601. [pathophysiological Pok] shock period
A, moving an open venous anastomosis, increased venous return
B, hyperaldosteronism, increased sodium and water reabsorption
C, anti-diuretic hormone increased, increase in water reabsorption
D, capacity vasoconstriction, increased venous return
E, ischemia and hypoxia increase erythropoiesis
Answer: D have a question here
Resolution: 194 602. [pathophysiological Pok] stage Ⅰ shock
A, capacity vasoconstriction, increased venous return
B, the capillary pressure drop, an increase in tissue fluid back
C, hyperaldosteronism, increased sodium and water reabsorption
D, anti-diuretic hormone increased, increase in water reabsorption
E, moving an open venous anastomosis, increased venous return
Answer: B here in doubt
resolution:
194603. [pathophysiological Pok] following shock is prone to DIC
A, septic shock
B, cardiogenic shock
C, anaphylactic shock
D, hemorrhagic shock
E, neurogenic shock
Answer: A point where there is doubt
Resolution: 194 604. [pathophysiological Pok] shock at the site of cell damage is the earliest
A, microsomes
B, mitochondria
C, lysosomal
D, high-thou matrix
E, cell membrane
Answer: E have questions here
resolution:
194605. [pathophysiological Pok] does not have the following humoral factor is the contraction of blood vessels
A, catecholamine
B, 5 – hydroxytryptamine
C, ET
D, atrial natriuretic peptide
E, angiotensin Ⅱ
Answer: D have a question here
Resolution: 194 606. [pathophysiological Pok] MODS is the most common cause of
A, malnutrition
B, severe trauma and infection
C, excessive infusion
D, oxygen concentration is too high
E, low immunity
Answer: B here in doubt
resolution:
194607. [pathophysiological Pok] the initial shock is due to the occurrence of acute renal failure
A, renal hypoperfusion
B, persistent renal ischemia
C, kidney toxin
D, acute tubular necrosis
E, ureteral obstruction
Answer: A point where there is doubt
Resolution: 194 608. [pathophysiological Pok] The following are the monitoring of shock fluid volume is the best indicator of
A, arterial blood pressure
B, heart rate
C, cardiac output
D, pulmonary artery wedge pressure
E, urine output
Answer: D have a question here
resolution:
194609. [pathophysiological Pok] selected vasodilator treatment of shock should first
A, to correct acidosis
B, to improve heart function
C, corticosteroids
D, the full expansion
E, given cytoprotective agent
Answer: D have a question here
Resolution: 194 610. [pathophysiological Pok] pH is abnormal
A, an increase in lactate production caused by ischemia and cellular pH reduction
B, removal of acidic products to reduce the ischemic tissue, pH lower
C, reperfusion of ischemic tissue quickly correct acidosis but will increase the cell damage
D, excessive use of basic drugs to the ischemic tissue acidosis alkalosis into
E, acidosis and alkalosis alternating
Answer: C There are questions here
resolution:
194611. [pathophysiological Pok] most prone to ischemia – reperfusion injury of the organ is
A, heart
B, liver
C, lung
D, kidney
E, gastrointestinal tract
Answer: A point where there is doubt
Resolution: 194 612. [pathophysiological Pok] xanthine dehydrogenase mainly in
A, vascular smooth muscle cells
B, vascular endothelial cells
C, cardiac myocytes
D, liver cells
E, white blood cells
Answer: B here in doubt
resolution:
194694. [pathophysiological Pok] The following are heart failure and pulmonary congestion performance is
A, jugular venous reflux and positive signs of liver
B, paroxysmal nocturnal dyspnea
C, lower extremity edema
D, tenderness, hepatomegaly
E, jugular vein distention
Answer: B here in doubt
Resolution: 194 695. [pathophysiological Pok] heart failure, the following compensatory response has lost its meaning is
A, heart rate
B, cardiac hypertrophy
C, the expansion of myogenic
D, polycythemia
E, blood flow redistribution
Answer: B here in doubt
resolution:
194696. [pathophysiological Pok] heart failure, the following compensatory response is caused primarily by the kidneys
A, polycythemia
B, blood flow redistribution
C, stress-induced expansion
D, increased myoglobin
E, an increase in the number of mitochondria
Answer: A point where there is doubt
Resolution: 194 697. [pathophysiological Pok] the concept of respiratory failure is not correct
A, respiratory failure due to severe respiratory disorders leading to outside PaO below normal or pathological process associated with the increase in PaCO
B, respiratory failure, blood gas to determine the standard is generally PaO 60mmHg, with or without PaCO> 50mmHg
C, respiratory failure can be divided into hypoxemia (Ⅰ type) and hypoxemia with hypercapnia type (Ⅱ)
D, patients with respiratory failure (without treatment) can only increase and not PaCO lower PaO
E, can be divided according to course through the different acute and chronic respiratory failure
Answer: D have a question here
resolution:
194698. [pathophysiological Pok] bronchial pneumonia as the main mechanism leading to hypoxemia
A, obstructive ventilatory
B, restrictive ventilatory defect
C, ventilation perfusion imbalance
D, diffusion barriers
E, increased pulmonary circuit
Answer: D have a question here
Resolution: 194 699. [pathophysiological Pok] patients, drowning suffocation, blood gas analysis by looting after the results of salvage: pH7.18, PaCO 9.9kPa (75mmHg), HCO 28mmol / L, the most likely acid base balance disorder type
A, metabolic acidosis
B, acute respiratory acidosis
C, chronic respiratory acidosis
D, metabolic acidosis with metabolic alkalosis
E, metabolic alkalosis
Answer: B here in doubt
resolution:
194700. [pathophysiological Pok] in patients with pyloric stenosis, the occurrence of repeated vomiting, blood gas analysis results: pH7.5, PaCO 6.6kPa (50mmHg), HCO 36mmol / L, the most likely acid-base balance disorder type
A, metabolic acidosis
B, metabolic alkalosis
C, respiratory acidosis
D, respiratory alkalosis
E, mixed alkalosis
Answer: B here in doubt
Resolution: 194 701. [pathophysiological Pok] long-term use of broad-spectrum antibiotics for bacterial diarrhea caused mostly
A, food poisoning
B, bacillary dysentery
C, allergic colitis
D, flora disorders
E, cholera-like diarrhea
Answer: D have a question here
resolution:
194702. [pathophysiological Pok] an important feature of phage
A, small size
B, was shaped or rod-shaped cones
C, can affect bacteria and protozoa
D, parasitic in living cells
E, simple structure, the capsid protein and nucleic acid composition of non-microbial cells
Answer: E have questions here
Resolution: 194 703. [pathophysiological Pok] so that the corresponding bacterial lysates of the phage is
A, moderate phage
B, phage toxicity
C, prophage
D, lysogenic phage
E, L bacterial
Answer: B here in doubt
resolution:
194704. [Pathophysiology of Pok] may determine the structural characteristics of bacterial resistance is
A, R plasmids
B, the cell membrane
C, metachromatic granules
D, cytoplasmic
E, nuclear and cytoplasmic
Answer: A point where there is doubt
Resolution: 194 705. [pathophysiological Pok] a bacterial cell growth in solid medium can be formed
A, biofilm
B, bacteria moss
C, colonies
D, mycelium
E, bacteria group
Answer: C There are questions here
resolution:
194706. [pathophysiological Pok] is a product of the bacterial catabolism
A, pigment
B, bacteriocin
C, vitamin
D, antibiotics
E, CO
Answer: E have questions here
Resolution: 194 707. [pathophysiological Pok] laboratory for drug sensitivity tests on bacteria, the best choice for bacterial growth period
A, slow phase
B, log phase
C, stable
D, recession
E, a period of dormancy
Answer: B here in doubt
resolution:
194708. [pathophysiological Pok] is a toxic component of endotoxin
A, protein A
B, lipid A
C, peptidoglycan
D, the core polysaccharide
E, specific polysaccharide
Answer: B here in doubt
Resolution: 194 709. [pathophysiological Pok] Bacillus Calmette-Guerin (BCG) from the
A, M. tuberculosis virulence variation occurs
B, tuberculosis in vivo activity of mutated
C, TB bacteria mutate structure
D, occurrence of antigenic variation of M. tuberculosis
E, TB mutation on drug sensitivity
Answer: A point where there is doubt
resolution:
194710. [pathophysiological Pok] pathogenic bacteria is mainly determined by
A, bacteria invade the portal
B, the strength of the body resistance
C, the number of bacteria invade the body
D, invasion of bacteria and bacterial toxins
E, the environmental factors surrounding the body
Answer: D have a question here
Resolution: 194 711. [pathophysiological Pok] is the meaning of sterilization
A, to kill pathogenic microorganisms on objects
B, prevention and inhibition of microbial growth
C, did not live all mean
D, control microbial growth
E, kill all the microorganisms on objects
Answer: E have questions here
resolution:
194712. [pathophysiological Pok] effective way to kill the bacteria Bacillus
A, dry heat sterilization
B, pasteurization
C, high-pressure steam sterilization
D, intermittent sterilization
E, boiling
Answer: C There are questions here
Resolution: 194 713. [pathophysiological Pok] is a common cause of hospital infections
A, Mycobacterium tuberculosis
B, Vibrio cholerae
C, Pseudomonas aeruginosa
D, Neisseria gonorrhoeae
E, Shigella dysenteriae
Answer: C There are questions here
resolution:
194763. [pathophysiological Pok] normal type anion gap metabolic acidosis seen in
A, severe diarrhea
B, mild renal failure
C, renal tubular acidosis
D, using carbonic anhydrase inhibitors
E, above all
Answer: E have questions here
Resolution: 194 764. [pathophysiological Pok] The following is not a cause of metabolic acidosis is
A, heat
B, shock
C, vomiting
D, diarrhea
E, hyperkalemia
Answer: C There are questions here
resolution:
194765. [pathophysiological Pok] chronic respiratory acidosis mainly by compensatory adjustment
A, respiratory compensation
B, cardiac decompensation
C, compensatory blood system
D, renal compensatory
E, skeletal compensatory
Answer: D have a question here
Resolution: 194 766. [pathophysiological curb] with the following
A, also known as blood doping
B, is part of the alveolar ventilation and blood flow did not significantly reduce the corresponding decrease in
C, the normal function of the shunt also
D, pulmonary vasoconstriction, also can cause functional shunt
E, functional shunt part of the arterial and venous blood can not be fully reduced PaCO increase in PaO
Answer: D have a question here
resolution:
194767. [pathophysiological Pok] is based on the pathophysiology of acute lung injury
A, a large number of activated white blood cells
B, extensive damage to alveolar endothelial cells
C, a wide range of alveolar capillary membrane damage
D, a large number of activated lung macrophages
E, acute pulmonary edema
Answer: C There are questions here
Resolution: 194 768. [pathophysiological Pok] the following double acid-base balance is unlikely
A, metabolic acidosis with respiratory alkalosis
B, metabolic acidosis with metabolic alkalosis
C, metabolic alkalosis with respiratory alkalosis
D, metabolic alkalosis with respiratory acidosis
E, respiratory acidosis with respiratory alkalosis
Answer: E have questions here
resolution:
194769. [pathophysiological Pok] affect arterial oxygen content of the main factors
A, cells the ability to deter oxygen
B, hemoglobin
C, arterial partial pressure of CO
D, arterial partial pressure of oxygen
E, erythrocyte 2,3-DPG levels
Answer: B here in doubt
Resolution: 194 770. [pathophysiological Pok] check move a venous oxygen content difference mainly reflects the
A, inhalation of gas partial pressure of oxygen
B, lung ventilation
C, the lung ventilation function
D, hemoglobin and oxygen affinity
E, organizations deter the ability to access and use of oxygen
Answer: E have questions here
resolution:
194771. [pathophysiological Pok] lead to hypoxia due to blood
A, cyanide poisoning
B, nitrite poisoning
C, sulfide poisoning
D, arsenic poisoning
E, methanol poisoning
Answer: B here in doubt
Resolution: 194 772. [pathophysiological Pok] hypoxia caused circulatory diseases
A, emphysema
B, anemia
C, arterial spasm
D, carbon monoxide poisoning
E, vitamin B deficiency
Answer: C There are questions here
resolution:
194783. [pathophysiological Pok] general adaptation syndrome in the period of resistance play a major role of the hormone is
A, adrenaline
B, norepinephrine
C, insulin
D, glucocorticoids
E, β-endorphin
Answer: D have a question here
Resolution: 194 784. [pathophysiological Pok] is a kind of stress ulcer
A, peptic ulcer
B, a post-traumatic superficial skin ulcers
C, serious illness, serious injury cases, the presence of gastric and duodenal mucosa of superficial ulcers
D, of psychological stress that occurs when oral ulcers
E, cancerous ulcer
Answer: C There are questions here
resolution:
194785. [pathophysiological Pok] to promote systemic shwartzman reactions occur because of DIC
A, synthetic anticoagulant barriers
B, hypercoagulability
C, monocytes – macrophages system dysfunction
D, microcirculation
E, inhibition of fibrinolysis
Answer: C There are questions here
Resolution: 194 786. [pathophysiological Pok] DIC onset usually
A, oliguria
B, bleeding
C, difficulty in breathing
D, anemia
E, drowsiness
Answer: B here in doubt
resolution:
194787. [pathophysiological Pok] DIC is caused by anemia
A, aplastic anemia
B, blood loss anemia
C, toxic anemia
D, hemolytic anemia
E, iron deficiency anemia
Answer: D have a question here
Resolution: 194788. [pathophysiological Pok] caused by microangiopathic hemolytic anemia is a major factor
A, a lot of damage to microvascular endothelial cells
B, fibrin strands form a fine network in the microvascular
C, blood stasis in small blood vessels
D, microvascular thrombosis in a large number of micro-
E, a strong contraction of small blood vessels
Answer: B here in doubt
resolution:
194789. [pathophysiological Pok] DIC, the performance of blood coagulation
A, increased coagulation
B, decreased coagulation
C, coagulation first increased and then decreased
D, coagulation increased after the first reduction
E, no significant changes in coagulation
Answer: C There are questions here
Resolution: 194 790. [pathophysiological Pok] extensive use of corticosteroids are likely to cause DIC is because
A, a large number of tissue thromboplastin into the blood
B, widespread damage to vascular endothelial cells
C, increased lysosomal membrane stability
D, mononuclear phagocyte system function suppression
E, reduced anticoagulant activity of heparin
Answer: D have a question here
resolution:
221664. [pathophysiological Pok] stasis of blood in the main deposition
A, arterioles and capillaries
B, small arteries and small veins
C, aortic and small artery
D, arterial and intracardiac
E, capillaries and small veins within the
Answer: E have questions here
Resolution: 318 256. [pathophysiological Pok] female, 36 years old, has been diagnosed with nephrotic syndrome, two days right leg pain, cold, touch right foot dorsal artery pulsatility clear, toe refers to the skin, cyanosis, should first complications is considered
A, lower urinary tract infection
B, right lower limb venous thrombosis
C, cardiogenic shock
D, acute renal failure
E, the right lower extremity arterial thrombosis
Answer: E have questions here
194773. [pathophysiological Pok] cyanide poisoning is characterized by changes in oxygen
A, reduced oxygen capacity
B, arterial oxygen content decreased
C, lower arterial oxygen pressure
D, Bao, and was reduced arterial oxygen
E, move – venous oxygen content difference lower
Answer: E have questions here
Resolution: 194 774. [pathophysiological Pok] erythrocytosis of chronic hypoxia is the mechanism
A, abdominal visceral vasoconstriction
B, liver and spleen release stored blood
C, reduced red blood cell destruction
D, an increase in hepatic erythropoietin
E, enhanced bone marrow
Answer: E have questions here
resolution:
194775. [pathophysiological Pok] The following description of the concept of heat is the correct one
A, 0.6 ℃ above normal body temperature
B, the process of heat production exceeds heat dissipation process
C, is a common disease
D, the body temperature regulation center point upward rise of body temperature
E, by the central regulation of body temperature regulation dysfunction caused by elevated body temperature
Answer: D have a question here
Resolution: 194 776. [pathophysiological Pok] The following is the body temperature is overheated
A, pre-menstrual women
B, women during pregnancy
C, after strenuous exercise
D, congenital absence of sweat glands
E, meningococcal meningitis
Answer: D have a question here
resolution:
194777. [pathophysiological Pok] infusion reactions occur in most of its causes fever is due to
A, allergy
B, drug toxicity
C, exotoxin contamination
D, endotoxin contamination
E, fungal contamination
Answer: D have a question here
Resolution: 194 778. [pathophysiological Pok] following production and release of leukocyte pyrogen is the largest number of cells
A, neutrophils
B, monocytes
C, eosinophils
D, hepatic stellate cells
E, lymphocyte
Answer: B here in doubt
resolution:
194779. [pathophysiological Pok] Endotoxin is
A, Gram-positive bacteria, bacteria-wall components, the active ingredient is a lipopolysaccharide
B, Gram-negative bacteria-wall components of bacteria, the active ingredient is a lipopolysaccharide
C, Gram-positive bacteria, bacteria-wall components, the active ingredient is the core polysaccharide
D, Gram-negative bacteria-wall components of bacteria, the active ingredient is the core polysaccharide
E, Gram-negative bacteria-wall components of bacteria, the active ingredient is a small molecule protein
Answer: B here in doubt
Resolution: 194 780. [pathophysiological Pok] temperature rise of the thermal characteristics of the metabolic
A, heat production and heat balance
B, heat is greater than the heat
C, heat production is greater than the heat
D, heat barriers
E, thermal barrier
Answer: B here in doubt
resolution:
194781. [pathophysiological Pok] outside of pyrogen induced fever is mainly
A, activation of local vascular endothelial cells, the release of proinflammatory substances
B, stimulate local nerve endings, the release of neurotransmitters
C, directly on the hypothalamic thermoregulatory center
D, production EP cells led to activation of endogenous pyrogen production and release of
E, accelerated catabolism, increased heat production
Answer: D have a question here
Resolution: 194 782. [pathophysiological Pok] general adaptation syndrome in the awareness phase play a major role of the hormone is
A, CRH
B, catecholamines
C, insulin
D, glucocorticoids
E, β-endorphin
Answer: B here in doubt
resolution:
194783. [pathophysiological Pok] general adaptation syndrome in the period of resistance play a major role of the hormone is
A, adrenaline
B, norepinephrine
C, insulin
D, glucocorticoids
E, β-endorphin
Answer: D have a question here
Resolution: 194 784. [pathophysiological Pok] is a kind of stress ulcer
A, peptic ulcer
B, a post-traumatic superficial skin ulcers
C, serious illness, serious injury cases, the presence of gastric and duodenal mucosa of superficial ulcers
D, of psychological stress that occurs when oral ulcers
E, cancerous ulcer
Answer: C There are questions here
resolution:
194785. [pathophysiological Pok] to promote systemic shwartzman reactions occur because of DIC
A, synthetic anticoagulant barriers
B, hypercoagulability
C, monocytes – macrophages system dysfunction
D, microcirculation
E, inhibition of fibrinolysis
Answer: C There are questions here
Resolution: 194 786. [pathophysiological Pok] DIC onset usually
A, oliguria
B, bleeding
C, difficulty in breathing
D, anemia
E, drowsiness
Answer: B here in doubt
resolution:
194787. [pathophysiological Pok] DIC is caused by anemia
A, aplastic anemia
B, blood loss anemia
C, toxic anemia
D, hemolytic anemia
E, iron deficiency anemia
Answer: D have a question here
Resolution: 194788. [pathophysiological Pok] caused by microangiopathic hemolytic anemia is a major factor
A, a lot of damage to microvascular endothelial cells
B, fibrin strands form a fine network in the microvascular
C, blood stasis in small blood vessels
D, microvascular thrombosis in a large number of micro-
E, a strong contraction of small blood vessels
Answer: B here in doubt
resolution:
194789. [pathophysiological Pok] DIC, the performance of blood coagulation
A, increased coagulation
B, decreased coagulation
C, coagulation first increased and then decreased
D, coagulation increased after the first reduction
E, no significant changes in coagulation
Answer: C There are questions here
Resolution: 194 790. [pathophysiological Pok] extensive use of corticosteroids are likely to cause DIC is because
A, a large number of tissue thromboplastin into the blood
B, widespread damage to vascular endothelial cells
C, increased lysosomal membrane stability
D, mononuclear phagocyte system function suppression
E, reduced anticoagulant activity of heparin
Answer: D have a question here
resolution:
221664. [pathophysiological Pok] stasis of blood in the main deposition
A, arterioles and capillaries
B, small arteries and small veins
C, aortic and small artery
D, arterial and intracardiac
E, capillaries and small veins within the
Answer: E have questions here
Resolution: 318 256. [pathophysiological Pok] female, 36 years old, has been diagnosed with nephrotic syndrome, two days right leg pain, cold, touch right foot dorsal artery pulsatility clear, toe refers to the skin, cyanosis, should first complications is considered
A, lower urinary tract infection
B, right lower limb venous thrombosis
C, cardiogenic shock
D, acute renal failure
E, the right lower extremity arterial thrombosis
Answer: E have questions here
194783. [pathophysiological Pok] general adaptation syndrome in the period of resistance play a major role of the hormone is
A, adrenaline
B, norepinephrine
C, insulin
D, glucocorticoids
E, β-endorphin
Answer: D have a question here
Resolution: 194 784. [pathophysiological Pok] is a kind of stress ulcer
A, peptic ulcer
B, a post-traumatic superficial skin ulcers
C, serious illness, serious injury cases, the presence of gastric and duodenal mucosa of superficial ulcers
D, of psychological stress that occurs when oral ulcers
E, cancerous ulcer
Answer: C There are questions here
resolution:
194785. [pathophysiological Pok] to promote systemic shwartzman reactions occur because of DIC
A, synthetic anticoagulant barriers
B, hypercoagulability
C, monocytes – macrophages system dysfunction
D, microcirculation
E, inhibition of fibrinolysis
Answer: C There are questions here
Resolution: 194 786. [pathophysiological Pok] DIC onset usually
A, oliguria
B, bleeding
C, difficulty in breathing
D, anemia
E, drowsiness
Answer: B here in doubt
resolution:
194787. [pathophysiological Pok] DIC is caused by anemia
A, aplastic anemia
B, blood loss anemia
C, toxic anemia
D, hemolytic anemia
E, iron deficiency anemia
Answer: D have a question here
Resolution: 194788. [pathophysiological Pok] caused by microangiopathic hemolytic anemia is a major factor
A, a lot of damage to microvascular endothelial cells
B, fibrin strands form a fine network in the microvascular
C, blood stasis in small blood vessels
D, microvascular thrombosis in a large number of micro-
E, a strong contraction of small blood vessels
Answer: B here in doubt
resolution:
194789. [pathophysiological Pok] DIC, the performance of blood coagulation
A, increased coagulation
B, decreased coagulation
C, coagulation first increased and then decreased
D, coagulation increased after the first reduction
E, no significant changes in coagulation
Answer: C There are questions here